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Covid-19 Far More Likely to Bond to Cells than SARS Due to HIV-Like Mutation?


Jon Schultz

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Thanks for the great Rense interview tonight, Dr. Niman. A few questions I hope you can find the time to comment on before too long:

1) Have any sequences of recently announced cases in Iran and Italy, especially been released? From the most recent sequences you have seen would you say that there are two strains or lineages now, the original one and the one with changes in the ORF8 gene? And is it known how their effects differ and how prevalent each is?

2) Why do you think the number of reported new cases in Chinese cities outside Hubei province has been markedly going down, rather than up? Do you think the Chinese government may be misreporting or purposely not testing because they think the damage caused by people not going to their jobs will be worse than the additional damage if they do?

3) Have you heard whether a cytokine storm and/or secondary bacterial infections are prevalent in Covid-19 pneumonia? If secondary bacterial infections are occurring in a fair number of cases, do you think it might be advisable for people to get a pneumovax vaccine? (I know you are not a medical doctor and cannot give medical advice, but hopefully you can safely share your thoughts.)

4) Any thoughts on the paper this story covers?

https://www.scmp.com/news/china/society/article/3052495/coronavirus-far-more-likely-sars-bond-human-cells-scientists-say

Thanks in advance!

 

Edited by Jon Schultz
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